Fauci Funded then Covered Up H7N9 Fabricated Virus in 2013
When we look at the sequence of events followed By Fauci the follow me I am science man, we see a distinct timeline between experiments designed to produce super viruses first spreading from birds to humans, then easily spread from human to human. We also see experiments designed to immunize hosts, masking symptoms in vaccinated people who are used as living incubation chambers for viruses to mutate and infect unvaccinated people with a more severe virus strain with higher fatality rates. Studies in 2013 through 2017 show a direct parallel with results we have seen in real life in 2021 heading into 2022.
2011 Oct
Ocular Tropism of Influenza A Viruses: Identification of H7 Subtype-Specific Host Responses in Human Respiratory and Ocular Cells
To study virus-host interactions in ocular tissue, we infected primary human corneal and conjunctival epithelial cells with H7, H5, and H1 subtype viruses. We found that numerous virus subtypes were capable of infecting and replicating in multiple human ocular cell types, with the highest titers observed with highly pathogenic H7N7 and H5N1 viruses.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3196405/
As we can see, as far back as 2011 scientists have been experimenting with super spreader viruses by mixing a series of viruses until they developed a man made concoction of bird flu viruses and mammal, or human viruses. The studies at first appeared to be designed to gather data. Scientists stretched the boundaries of imagination in an attempt to first imagine, then create the worst case virus scenario able to spread from species to species, host to host, overcome vaccines, and spread through vaccination attempts. Is it by chance Fauci has been able to predict courses covid-19 has taken, or is Dr Fauci revealing bits and pieces of what scientists have learned from the creation and release of those super viruses in the name of science and research.
December 30, 2011
By Anthony S. Fauci
A deadly influenza virus has circulated widely in birds in recent years, decimating flocks but rarely spreading to humans. Nonetheless, because of its persistence in bird flocks, this highly pathogenic avian influenza virus has loomed as a major public health threat. Seasonal influenza kills less than 1 percent of the people it infects. In contrast, human infections with H5N1, though exceedingly rare, are fatal in more than half of cases. Should this virus mutate in a way that allows it to be transmitted as efficiently among people as seasonal influenza viruses are, it could take an unprecedented toll on human life.
A number of important scientific and public health questions regarding this virus remain unanswered, including the likelihood of such mutations arising and the mechanisms by which they may occur. Two recent studies co-funded by the National Institutes of Health have shed light on how this potentially grave human health threat could become a reality. Working carefully with influenza viruses they have engineered in isolated biocontainment laboratories, scientists in Europe and the United States have identified several mechanisms by which the virus might evolve to transmit efficiently in the ferret, the best animal model for human influenza infection.
Given these uncertainties, important information and insights can come from generating a potentially dangerous virus in the laboratory.
The question is whether benefits of such research outweigh risks. The answer is not simple. A highly pathogenic bird flu virus transmissible in humans could arise in ways not predicted by laboratory studies. And it is not clear whether this laboratory virus would behave in humans as it does in ferrets. Nonetheless, new data provide valuable insights that can inform influenza preparedness and help delineate the principles of virus transmission between species.
In a letter designed to gain funding for his experiments, Dr Fauci shares insight to the goals of those experiments. Since it was nearly impossible for bird flu to transmit to humans and survive in humans, Dr Fauci developed a set of experiments to first develop a new super virus capable of creating a bridge between bird and humans. Then a series of experiments to study how quickly and in how many different ways that new man made virus would be capable of spreading from human to human, country to country, then how a new super virus could overcome vaccinations, mutate, and spread. To say the least, the release of Dr Fauci’s new super virus was a super form of job security with nearly unlimited funding.
Sunday, 28 April 2013 07:38 PM
Anthony Fauci: New Bird Flu Virus Could Spread Among Humans
There is no evidence that the deadly H7N9 bird flu has yet spread between humans in China but health authorities must be ready for the virus to mutate at any time, a top US virologist has warned.
Anthony Fauci, the head of the National Institute of Allergy and Infectious Diseases (NIAID), said officials in China had studied more than 1,000 close contacts of confirmed cases and not found any evidence of human-to-human transmission.
“That is powerful evidence because if you had a thousand contacts with someone with the flu you would be pretty sure some of them would have been infected,” Fauci said in an interview with AFP.
https://www.newsmax.com/US/fauci-bird-flu-human/2013/04/28/id/501718/
In April of 2013 we see the first suspected public release of Fauci’s super virus. The virus was able to infect 1000 people in China, but Fauci voiced concern when the results showed the virus was unable to spread from human to human.
May 8, 2013 2:49 AM ET
Officials Prepare For Another Flu Pandemic — Just In Case
There’s been a buzz of activity at the Centers for Disease Control and Prevention in Atlanta since scientists got their first samples of a new bird flu virus from China four weeks ago.
Already they’ve prepared “seed strains” of the virus, called H7N9, and distributed them to vaccine manufacturers so the companies can grow them up and make them into experimental flu vaccine.
They’ve also come up with a new diagnostic test for H7N9 that the Food and Drug Administration has approved on an emergency basis.
Meanwhile, CDC researchers have almost concluded experiments that involve infecting ferrets with H7N9 — in particular, a virus derived from a 35-year-old Chinese housewife who died last Monday from the flu.
Because ferrets’ respiratory tracts are similar to those in humans, researchers want to see if the animals can infect each other through droplets in their exhalations, a possible clue to the all-important issue of whether this virus has the potential to spread readily among humans.
Dr. Daniel Jernigan, deputy director of CDC’s influenza division, says there are two main reasons why H7N9 is getting so much attention.
First, people who get it usually get very sick, and so far nearly a quarter of the 130 human cases have resulted in death.
Second, the genetic makeup of this new virus is disturbingly different from an older bird flu virus that has sickened more than 600 people over the past 10 years and killed more than half of them. That virus is an H5 type.
“The thing that’s different between them is the H5 virus still maintains a lot of the avian or bird flu characteristics,” Jernigan tells Shots, “whereas this H7N9 shows some adaptation to mammals. And that’s what makes it different and concerning for us.”
For instance, the newer bird flu has adapted to thrive at human body temperature, which is much lower than the body temperature of birds. It’s a necessary step toward becoming a flu that could spread easily from human to human — but it’s not sufficient.
One month after reviewing results from the first public information on the new super virus detected in China in April 2013, laboratories experimented with a new strain in ferrets that showed promises of spreading from ferret to ferret.
2013 Jul-Aug
H7N9 Avian Influenza A Virus and the Perpetual Challenge of Potential Human Pandemicity
The ongoing H7N9 influenza epizootic in China once again presents us questions about the origin of pandemics and how to recognize them in early stages of development. Over the past ~135 years, H7 influenza viruses have neither caused pandemics nor been recognized as having undergone human adaptation. Yet several unusual properties of these viruses, including their poultry epizootic potential, mammalian adaptation, and atypical clinical syndromes in rarely infected humans, suggest that they may be different from other avian influenza viruses, thus questioning any assurance that the likelihood of human adaptation is low.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3705455/
Science was unable to study bird flu in humans because cases were so rare for 135 years. Developing a new man made strain of bird flu in the laboratory was the only solution.
07 August 2013
Gain-of-function experiments on H7N9
To better assess the pandemic threat posed by A(H7N9) viruses, investigators from the NIAID Centers of Excellence in Influenza Research and Surveillance and other expert laboratories in China and elsewhere have characterized the wild-type avian A(H7N9) viruses in terms of host range, virulence and transmission, and are evaluating the effectiveness of antiviral drugs and vaccine candidates. However, to fully assess the potential risk associated with these novel viruses, there is a need for further research, including experiments that may be classified as ‘gain of function’ (GOF).
Normally, epidemiological studies and characterization of viruses from field isolates are used to inform policy decisions regarding public-health responses to a potential pandemic. However, classical epidemiological tracking does not give public-health authorities the time they need to mount an effective response to mitigate the effects of a pandemic virus. To provide information that can assist surveillance activities — thus enabling appropriate public-health preparations to be initiated before a pandemic — experiments that may result in GOF are critical.
Therefore, after review and approval, we propose to perform experiments that may result in GOF (see ‘Proposed gain-of-function experiments’).
https://www.nature.com/articles/500150a
Fauci sought funding for Gain of Function experiments which simply developed new strains of viruses for experiment purposes. The threat of a worldwide pandemic with loss of human life was a small price to pay for the information scientists hoped to gather. When we put timelines together we see how developments in laboratories are generally followed by limited outbreaks of the virus in small urban populations in China. Not once, but a series of outbreaks within months of every major break through in laboratories.
Aug 8, 2013,08:43am EDT
In an outrageous display of chutzpah, a group of flu researchers led by Ron Fouchier of Erasmus Medical Center in the Netherlands announced today, in a letter to the journal Nature, that they were planning to engineer the new H7N9 avian flu strain to give it new, possibly much more deadly capabilities. Fouchier is the same scientist who, two years ago, adapted the highly pathogenic H5N1 flu strain so that it could be passed from human to human, which it cannot do in its natural form. The resulting outcry delayed publication of his paper, but it eventually did appear.
Although Fouchier is in Rotterdam, the NIH funds part of his work through the National Institute of Allergies and Infectious Diseases (NIAID). Dr. Anthony Fauci, the head of NIAID, offered the reassurance that a special panel will review this H7N9 project, and
“If the risk is felt to be too high by this outside review, they will recommend it won’t be done and we won’t fund it.”
Some scientists shared their thoughts and plans. If we had paid better attention to the warning signs in 2013, everything we’ve seen in 2020 and 2021 could have been avoided. When research was made public, Dr Fauci simply assured the public, funding would be cut if experiments got out of hand. So far all we’ve seen is viruses spreading faster, more serious symptoms, and Fauci receiving unlimited funding.
2014 Sep 25
Pandemic potential of H7N9 influenza viruses
Avian influenza viruses rarely infect humans, but the recently emerged avian H7N9 influenza viruses have caused sporadic infections in humans in China, resulting in 440 confirmed cases with 122 fatalities as of May 16, 2014. In addition, epidemiologic surveys suggest that there have been asymptomatic or mild human infections with H7N9 viruses. These viruses replicate efficiently in mammals, show limited transmissibility in ferrets and guinea pigs, and possess mammalian-adapting amino acid changes that likely contribute to their ability to infect mammals. Here, we summarize the characteristic features of the novel H7N9 viruses and assess their pandemic potential.
Despite the wide host range of influenza A viruses, their transmission from avian to mammalian species or vice versa is rare due to host range restrictions. Influenza A viruses circulating in avian species (so-called ‘avian influenza viruses’) rarely infects humans, and influenza A viruses circulating in humans (‘human influenza viruses’) rarely infect avian species 4–7. Recently, however, avian influenza A viruses of the H5N1 and H7N9 subtypes have caused hundreds of cases of human infections. So far, sustained human-to-human transmission of these viruses has not been reported. Nonetheless, additional adaptive mutations and/or reassortment with circulating human viruses may enable H5N1 or H7N9 viruses to efficiently infect humans and transmit among them. Because humans lack protective antibodies against these viruses, human-transmitting H5N1 or H7N9 viruses could spread worldwide, resulting in an influenza pandemic.
Human infections with avian H7N9 influenza viruses in China in 2013–2014
To date, two sizable waves of human infection with H7N9 viruses have been documented (Figure 2A). The first wave started with a human case of H7N9 influenza virus infection in Shanghai on February 19, 2013 (this case was officially reported on March 31, 2013) 8. In April 2013, the number of human cases of H7N9 virus infections increased significantly, reaching 125 confirmed cases in China by the end of April.
Epidemiological studies have shown that H7N9 virus infections have affected mainly middle-aged or older individuals (Figure 2C; i.e., the median age at infection is 63 years) 19–29. Interestingly, two-thirds of the infected individuals have been male 19, 20, 22–24, 27–29 (Figure 2C). The high number of cases among elderly men may reflect socio-economical differences among age groups and genders since elderly men may have frequent work-related or non-job-related contact with poultry. Most H7N9 influenza patients exhibit general influenza-like symptoms, including fever and cough, and more than half of the infections typically progress to severe pneumonia, acute respiratory distress syndrome (ARDS), and multi-organ failure 8, 11, 19, 21, 23, 25, 27, 30–38. Most H7N9 virus-infected patients possessed at least one underlying medical condition, such as chronic obstructive pulmonary disease (COPD), diabetes, hypertension, obesity, and/or chronic lung and heart disease 19, 21–23, 25, 26, 28, 31, 39, 40, suggesting that these comorbidities may increase the risk of severe H7N9 virus infection.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4252989/
In 2014 bird flu rarely infected humans and it was impossible for the virus to spread from human to human. Scientists were convinced there was a possibility of the virus eventually spreading from human to human and feared the results. Keep in mind, the H7N9 viruses are man made, and would have never posed a treat if they weren’t somehow released to the public in China. Scientists continued studies to develop a strain that could transmit from human to human.
2014 Dec 8
Gain-of-function experiments: time for a real debate
According to the WHO, dual use research of concern (DURC) is “life sciences research that is intended for benefit, but which might easily be misapplied to do harm”. Recent studies, particularly those on influenza viruses, have led to renewed attention on DURC, as there is an ongoing debate over whether the benefits of gain-of-function (GOF) experiments that result in an increase in the transmission and/or pathogenicity of potential pandemic pathogens (PPPs) are outweighed by concerns over biosecurity and biosafety. In this Viewpoint article, proponents and opponents of GOF experiments discuss the benefits and risks associated with these studies, as well as the implications of the current debate for the scientific community and the general public, and suggest how the current discussion should move forward.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7097416/
Studies and debates continued through the end of 2014 justifying the development of a bird flu transmitted to humans then easily transmitted from human to human. All in the name of science.
2015 Jul; 13
Imperfect Vaccination Can Enhance the Transmission of Highly Virulent Pathogens
Could some vaccines drive the evolution of more virulent pathogens? Conventional wisdom is that natural selection will remove highly lethal pathogens if host death greatly reduces transmission. Vaccines that keep hosts alive but still allow transmission could thus allow very virulent strains to circulate in a population. Here we show experimentally that immunization of chickens against Marek’s disease virus enhances the fitness of more virulent strains, making it possible for hyperpathogenic strains to transmit. Immunity elicited by direct vaccination or by maternal vaccination prolongs host survival but does not prevent infection, viral replication or transmission, thus extending the infectious periods of strains otherwise too lethal to persist. Our data show that anti-disease vaccines that do not prevent transmission can create conditions that promote the emergence of pathogen strains that cause more severe disease in unvaccinated hosts.
Infectious agents can rapidly evolve in response to health interventions [1]. Here, we ask whether pathogen adaptation to vaccinated hosts can result in the evolution of more virulent pathogens (defined here to mean those that cause more or faster mortality in unvaccinated hosts).
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4516275/
Continued studies revealed, the use of a faulty vaccine was the key to spreading the man made H7N9 virus from human to human. These studied also showed, vaccinated individuals were able to spread that man made virus to unvaccinated individuals. A goal Dr Fauci wrote about in 2011 and 2013.
2016 Aug 8
Gain-of-Function Research: Ethical Analysis
Gain-of-function (GOF) research involves experimentation that aims or is expected to (and/or, perhaps, actually does) increase the transmissibility and/or virulence of pathogens. Such research, when conducted by responsible scientists, usually aims to improve understanding of disease causing agents, their interaction with human hosts, and/or their potential to cause pandemics. The ultimate objective of such research is to better inform public health and preparedness efforts and/or development of medical countermeasures. Despite these important potential benefits, GOF research (GOFR) can pose risks regarding biosecurity and biosafety. GOFR is a subset of “dual-use research”—i.e., research that can be used for both beneficial and malevolent purposes. Whereas the dual-use life science research debate has largely focused on biosecurity dangers associated with potential malevolent use of research, the GOFR debate has more explicitly focused on risks involving both biosecurity and biosafety—the point being that creation of especially dangerous pathogens might pose highly significant biosafety risks that are independent of, and perhaps more feasible to measure/assess than, risks associated with malevolent use.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4996883/
As the debate raged, scientists who knew a super virus was released to the public argued for additional funds to study the mutation within humans, the spread, and role vaccines would play in the spread of a new super virus. All of this man made. Funding was needed, the gloves were off, and scientists knew the plague was out of the box.
2017 Dec 1
Genesis and Spread of Newly Emerged Highly Pathogenic H7N9 Avian Viruses in Mainland China
The novel low-pathogenic avian influenza A H7N9 viruses (LPAI H7N9 viruses) have been a threat to public health since their emergence in 2013 because of the high rates of mortality and morbidity that they cause. Recently, highly pathogenic variants of these avian influenza A H7N9 viruses (HPAI H7N9 viruses) have emerged and caused human infections and outbreaks among poultry in mainland China.
IMPORTANCE The LPAI H7N9 virus has caused five outbreak waves in humans and was recently reported to have mutated into highly pathogenic variants. It is unknown how the HPAI H7N9 virus originated, evolved, and disseminated in China.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5686710/
After five outbreaks in China over a four year period, warnings were finally issued in December 2017. Science was at a loss to combat this new man made super virus. in 2014 it was rare for bird flu to jump to humans, and impossible for the virus to jump from human to human. Laboratory experiments over that four year period developed a strain able to transmit from human to human. Additional experiments showed how a faulty vaccine could help spread the virus while being unsuccessful at controlling the virus. All this information was known in 2017. Two years later we saw the release of this super virus known as covid-19. 2020 and 2021 have shown the results of those experiments. The deaths of hundreds of thousands of people, a continued mutation of the virus, and a virus designed to work with vaccines to increase the spread of the virus. Since 2011 Dr Fauci has been securing funds for experiments directly related to the refinement and spread of the worst feared virus. Today we are not only seeing the effects of those experiments, we have become the unwilling and misinformed subjects of those experiments.